ALOPECIA-do dr.JARKA
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AGA I IRYD
- Posty:58
- Rejestracja:25 września 2007, 22:22
- Lokalizacja:Częstochowa
Mam pytanko odnośnie tego schorzenia.Pojawia się głównie u ras "szpicowych".Przesłano mi informacje obcojęzyczne na temat tej choroby.Objawy całkowicie przypominają chorobe mojego psa.Czy można gdzies w Polsce przeprowadzić testy na obecnośc tej choroby,za oceanem koszt to ok 100 dolarów.Nie chodzi mi o fundusze,ale o pomoc w przedłużeniu życia mojemu psu.W linku jest mowa o leku TRILOSTANE i zmianie diety (łosoś i ziemniaki).Podobno choroba powoduje szybkie starzenie całego organizmu psa i choroby nerek.Proszę o pomoc.AGA I IRYD....
A czy to na pewno ta choroba? Alopecia to łysienie. Tutaj masz opis ale po angielsku 
Alopecia is the absence of hair; hypotrichosis, which is much more common, is the presence of less hair than normal. Although these defects can be generalized, they commonly develop in patterns that spare the extremities or correlate with hair color. These ectodermal defects can be congenital or tardive and can be associated with abnormal or absent adnexa, with defects in other ectodermal structures (such as teeth, claws, and eyes), or with skeletal and other developmental defects. There are various modes of inheritance in those instances in which familial occurrence has been studied. X-linked ectodermal dysplasia has been recently reported in German Shepherds. Hairless breeds of dogs (eg, Mexican Hairless, Chinese Crested, American Hairless Terrier) and cats (Sphinx) have been bred for these ectodermal defects. Many sporadic cases of ectodermal defects are described in dogs, most often in males. Many affected dogs, including most of the hairless breeds, have patchy or pattern hypotrichosis as well as associated dental anomalies. All animals with abnormal follicular development are prone to comedone formation, hair follicle infections, and hair foreign-body granulomas.
At least 13 types of hypotrichosis have been described in cattle, affecting Angus; Ayrshire; Brangus; Holstein-Friesian; Hereford; polled Hereford; Guernsey; Gelbvieh; Jersey; and Normandy-Maine, Anjou-Charolais, and Simmental crosses. Most have autosomal recessive or sex-linked modes of inheritance. Associated defects include failure of horn development, hypophyseal hypoplasia, macroglossia, dental anomalies, abnormal coat coloration, and death (lethal hypotrichosis). Viable hypotrichosis, hypotrichosis with anodontia, semi-hairlessness, streaked hairlessness, black hair follicle dysplasia (Holstein), and cross-related hypotrichosis (rat tail) are specific types described in cattle.
In sheep, hypotrichosis is rarely reported, with the best known syndrome affecting the Polled Dorset. This involves the hair of the face most severely, but the wool is also of poor quality. In goats, hypotrichosis is associated with congenital goiter. In swine, 2 forms of hypotrichosis are known (Mexican Hairless, German), one of which is associated with goiter and death in the homozygote.
Photographs
Color dilution alopecia, Doberman
Color dilution alopecia, Doberman
In dogs, there are several tardive follicle dysplasias, including color dilution alopecia. This is found in some dogs bearing the coat color genotype dd, which renders black genotypes blue and liver genotypes beige or fawn. This syndrome is best known in Doberman Pinschers but is also commonly seen in color dilute Dachshunds, Italian Greyhounds, Greyhounds, Whippets, Yorkshire Terriers, and tricolor hounds and has now been reported in a German Shepherd. Affected dogs are born with normal hair coats but before 1 yr of age begin to develop folliculitis and hypotrichosis that is progressive and confined to the blue or fawn colored areas. Black hair follicle dysplasia, a similar but earlier developing and more complete hypotrichosis, is seen in black and white piebald dogs. The hypotrichosis develops shortly after birth and affects only the black-colored areas. This syndrome is best known in the Papillon and Bearded Collie. Recent genetic analysis in Large Munsterlanders has indicated an autosomal recessive inheritance in this breed. A similar follicular dysplasia is reported in nonpiebald breeds. Other types of follicular dysplasias that are apparently affected by endocrine and dietary factors are seasonal flank alopecia of Boxers and Airedale Terriers and various woolly syndromes and post-clipping alopecia in Spitz-type breeds. Familial hypotrichosis of Irish Water Spaniels has been shown to be influenced by both dietary factors and sex hormones. The condition formerly known as growth hormone-responsive alopecia in Pomeranians and other breeds is now called alopecia X, reflecting the complexity of factors, hereditary and otherwise, influencing these syndromes.
In cats, follicular dysplasia occurs in the Devon Rex. In horses, both color dilution alopecia and black hair follicle dysplasias are occasionally reported, especially in Appaloosas. Congenital progressive hypotrichosis has been reported in a blue roan Percheron. Reported hair shaft structural abnormalities of dogs and cats include pili torti (American Wirehaired Cat), trichorrhexis nodosa, and spiculosis (Kerry Blue Terrier).
Alopecia is the absence of hair; hypotrichosis, which is much more common, is the presence of less hair than normal. Although these defects can be generalized, they commonly develop in patterns that spare the extremities or correlate with hair color. These ectodermal defects can be congenital or tardive and can be associated with abnormal or absent adnexa, with defects in other ectodermal structures (such as teeth, claws, and eyes), or with skeletal and other developmental defects. There are various modes of inheritance in those instances in which familial occurrence has been studied. X-linked ectodermal dysplasia has been recently reported in German Shepherds. Hairless breeds of dogs (eg, Mexican Hairless, Chinese Crested, American Hairless Terrier) and cats (Sphinx) have been bred for these ectodermal defects. Many sporadic cases of ectodermal defects are described in dogs, most often in males. Many affected dogs, including most of the hairless breeds, have patchy or pattern hypotrichosis as well as associated dental anomalies. All animals with abnormal follicular development are prone to comedone formation, hair follicle infections, and hair foreign-body granulomas.
At least 13 types of hypotrichosis have been described in cattle, affecting Angus; Ayrshire; Brangus; Holstein-Friesian; Hereford; polled Hereford; Guernsey; Gelbvieh; Jersey; and Normandy-Maine, Anjou-Charolais, and Simmental crosses. Most have autosomal recessive or sex-linked modes of inheritance. Associated defects include failure of horn development, hypophyseal hypoplasia, macroglossia, dental anomalies, abnormal coat coloration, and death (lethal hypotrichosis). Viable hypotrichosis, hypotrichosis with anodontia, semi-hairlessness, streaked hairlessness, black hair follicle dysplasia (Holstein), and cross-related hypotrichosis (rat tail) are specific types described in cattle.
In sheep, hypotrichosis is rarely reported, with the best known syndrome affecting the Polled Dorset. This involves the hair of the face most severely, but the wool is also of poor quality. In goats, hypotrichosis is associated with congenital goiter. In swine, 2 forms of hypotrichosis are known (Mexican Hairless, German), one of which is associated with goiter and death in the homozygote.
Photographs
Color dilution alopecia, Doberman
Color dilution alopecia, Doberman
In dogs, there are several tardive follicle dysplasias, including color dilution alopecia. This is found in some dogs bearing the coat color genotype dd, which renders black genotypes blue and liver genotypes beige or fawn. This syndrome is best known in Doberman Pinschers but is also commonly seen in color dilute Dachshunds, Italian Greyhounds, Greyhounds, Whippets, Yorkshire Terriers, and tricolor hounds and has now been reported in a German Shepherd. Affected dogs are born with normal hair coats but before 1 yr of age begin to develop folliculitis and hypotrichosis that is progressive and confined to the blue or fawn colored areas. Black hair follicle dysplasia, a similar but earlier developing and more complete hypotrichosis, is seen in black and white piebald dogs. The hypotrichosis develops shortly after birth and affects only the black-colored areas. This syndrome is best known in the Papillon and Bearded Collie. Recent genetic analysis in Large Munsterlanders has indicated an autosomal recessive inheritance in this breed. A similar follicular dysplasia is reported in nonpiebald breeds. Other types of follicular dysplasias that are apparently affected by endocrine and dietary factors are seasonal flank alopecia of Boxers and Airedale Terriers and various woolly syndromes and post-clipping alopecia in Spitz-type breeds. Familial hypotrichosis of Irish Water Spaniels has been shown to be influenced by both dietary factors and sex hormones. The condition formerly known as growth hormone-responsive alopecia in Pomeranians and other breeds is now called alopecia X, reflecting the complexity of factors, hereditary and otherwise, influencing these syndromes.
In cats, follicular dysplasia occurs in the Devon Rex. In horses, both color dilution alopecia and black hair follicle dysplasias are occasionally reported, especially in Appaloosas. Congenital progressive hypotrichosis has been reported in a blue roan Percheron. Reported hair shaft structural abnormalities of dogs and cats include pili torti (American Wirehaired Cat), trichorrhexis nodosa, and spiculosis (Kerry Blue Terrier).
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AGA I IRYD
- Posty:58
- Rejestracja:25 września 2007, 22:22
- Lokalizacja:Częstochowa
Aby potwierdzić chorobę najlepiej wykonać testy.Znajoma zabiera próbki ( skóry i włosów) swoich psów do Szwajcari,aby wykluczyć chorobę w jej hodowli .Mój pies pochodzi z innej hodowli,jest to pierwszy miot po psie z Ukrainy.Wszystkie opisy choroby są podobne do wizualnego opisu mojego psa.Dodam ,Iryd jest rasy szpic wilczy.Ostatnio właśnie ta choroba zaczeła atakować tą rase.Najbardziej charakterystyczne u samców są wyłysienia na tylnych łapach,skóra zabarwia się na czarno.AGA...
samo słowo alopetia to łysienie, badania w kierunku łysienia szpiców wykonują zachodnie labolatoria. Nie miełaem jeszcze chętnego na takie badania więc więcej szczegułów nie znam.
cushing jest chorobą bardzo rzadką , ale posiadajaca bardzo typowe symptomy. Jedna z nich jest nadmierne picie i nadmierne sikanie , powiększony brzuch, obwisły, przerzedzony ostry nietypowy włos, scieńczała skóra. Mozna wykonywa prosty test test hamowania dexamethazonem.